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hela s3 cells  (ATCC)


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    ATCC hela s3 cells
    Hela S3 Cells, supplied by ATCC, used in various techniques. Bioz Stars score: 97/100, based on 1700 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/hela+ccl+2/HeLa+S3/us12655229-426-8-11
    Average 97 stars, based on 1700 article reviews
    hela s3 cells - by Bioz Stars, 2026-09
    97/100 stars

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    Related Articles

    other:

    Article Title: miR-19a-3p as a Candidate oncomiRNA in Cervical Cancer: Insights From a Bioinformatic Analysis
    Article Snippet: The SiHa (HTB-35), HeLa (CCL-2) and C-33A (HTB-31TM) cell lines were purchased from American Type Culture Collection (ATCC, Manassas, VA, USA).

    Article Title: Hierarchically Engineered Multi-Enzyme Nanoreactors for in vitro Drug Biosynthesis and Pathway Transplantation Into Cells.
    Article Snippet: HeLa (CCL-2) and MCF7 (HTB-22) cell lines were obtained from ATCC.

    Article Title: Molecular determinants of Bcl-xL membrane insertion – Structural plasticity exploration in solution and in nanodiscs identifies IsoAsp deamidation as a loss-of function mechanism in vivo
    Article Snippet: HeLa (CCL-2) and HCT116 (CCL-247) cells were purchased from the ATCC®.

    Article Title: Therapeutic implications of targeting cancer testis antigen MAGEA1 in cervical cancer
    Article Snippet: HeLa (CCL-2) and Panc1 (CRL-1469) were purchased from American Type Culture Collection.

    Article Title: Therapeutic implications of targeting cancer testis antigen MAGEA1 in cervical cancer.
    Article Snippet: HeLa (CCL‐2) and Panc1 (CRL‐1469) were purchased from American Type Culture Collection.

    Cell Culture:

    Article Title: A bidirectional interaction between the SREBP pathway and the LINC complex component nesprin-4 controls lipid metabolism
    Article Snippet: .. MCF7 (HTB-22), HepG2-C3A (CRL-10741), HeLa (CCL-2) and HEK293 (CRL-1573) cells were obtained from American Type Cell Culture Collection (ATCC). .. Cell culture media, supplements and reagents were from Gibco.

    Modification:

    Article Title: MDS/AML-associated DDX41 helicase facilitates homologous recombination repair by potentially resolving R-loops
    Article Snippet: .. HeLa (CCL-2) and HT1080 (CCL-121, both from ATCC) cells were grown in Dulbecco’s modified Eagle medium (DMEM, 11995065, Gibco) supplemented with 10% fetal bovine serum (F1051, Sigma) and penicillin/streptomycin (100 U/ml each, P4333, Sigma). .. U2OS (HTB-96, ATCC) and U2OS GFP reporter cells [ ] (a gift from Jeremy Stark, City of Hope) were grown in Mccoy 5A media (16600082, Thermo Fisher) supplemented with 10% fetal bovine serum.

    Article Title: MDS/AML-associated DDX41 helicase facilitates homologous recombination repair by potentially resolving R-loops.
    Article Snippet: .. HeLa (CCL-2) and HT1080 (CCL-121, both from ATCC) cells were grown in Dulbecco’s modified Eagle medium (DMEM, 11995065, Gibco) supplemented with 10% fetal bovine serum (F1051, Sigma) and penicillin/streptomycin (100 U/ml each, P4333, Sigma). .. U2OS (HTB-96, ATCC) and U2OS GFP reporter cells [ 83 ] (a gift from Jeremy Stark, City of Hope) were grown in Mccoy 5A media (16600082, Thermo Fisher) supplemented with 10% fetal bovine serum.



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    ATCC hela s3 cells
    Hela S3 Cells, supplied by ATCC, used in various techniques. Bioz Stars score: 97/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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    Hypoxia increases expression of EZH2, H3K27me3 and survivin. (A) Immunoblots of WCEs <t>from</t> <t>U2OS,</t> <t>HeLa</t> and MRC5 lines cultured under normoxic or hypoxic environments (24 h). Blots were immunoprobed with anti-EZH2, anti-H3K27me3 and anti-survivin antibodies. Anti-Hif1a used to prove the hypoxic state had been induced, and anti-tubulin was used as a loading control. (B–D) Quantification of immunoblots represented in A from three independent experiments demonstrating that EZH2, H3K27me3 and survivin are all more abundant under hypoxia. Data presented are means±s.d. * P <0.05, ** P <0.01, *** P <0.001 (two-way ANOVA with Tukey's multiple comparisons post test).
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    Hypoxia increases expression of EZH2, H3K27me3 and survivin. (A) Immunoblots of WCEs <t>from</t> <t>U2OS,</t> <t>HeLa</t> and MRC5 lines cultured under normoxic or hypoxic environments (24 h). Blots were immunoprobed with anti-EZH2, anti-H3K27me3 and anti-survivin antibodies. Anti-Hif1a used to prove the hypoxic state had been induced, and anti-tubulin was used as a loading control. (B–D) Quantification of immunoblots represented in A from three independent experiments demonstrating that EZH2, H3K27me3 and survivin are all more abundant under hypoxia. Data presented are means±s.d. * P <0.05, ** P <0.01, *** P <0.001 (two-way ANOVA with Tukey's multiple comparisons post test).
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    Hypoxia increases expression of EZH2, H3K27me3 and survivin. (A) Immunoblots of WCEs <t>from</t> <t>U2OS,</t> <t>HeLa</t> and MRC5 lines cultured under normoxic or hypoxic environments (24 h). Blots were immunoprobed with anti-EZH2, anti-H3K27me3 and anti-survivin antibodies. Anti-Hif1a used to prove the hypoxic state had been induced, and anti-tubulin was used as a loading control. (B–D) Quantification of immunoblots represented in A from three independent experiments demonstrating that EZH2, H3K27me3 and survivin are all more abundant under hypoxia. Data presented are means±s.d. * P <0.05, ** P <0.01, *** P <0.001 (two-way ANOVA with Tukey's multiple comparisons post test).
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    ATCC hela 229 cell
    Hypoxia increases expression of EZH2, H3K27me3 and survivin. (A) Immunoblots of WCEs <t>from</t> <t>U2OS,</t> <t>HeLa</t> and MRC5 lines cultured under normoxic or hypoxic environments (24 h). Blots were immunoprobed with anti-EZH2, anti-H3K27me3 and anti-survivin antibodies. Anti-Hif1a used to prove the hypoxic state had been induced, and anti-tubulin was used as a loading control. (B–D) Quantification of immunoblots represented in A from three independent experiments demonstrating that EZH2, H3K27me3 and survivin are all more abundant under hypoxia. Data presented are means±s.d. * P <0.05, ** P <0.01, *** P <0.001 (two-way ANOVA with Tukey's multiple comparisons post test).
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    ATCC hela s3
    Hypoxia increases expression of EZH2, H3K27me3 and survivin. (A) Immunoblots of WCEs <t>from</t> <t>U2OS,</t> <t>HeLa</t> and MRC5 lines cultured under normoxic or hypoxic environments (24 h). Blots were immunoprobed with anti-EZH2, anti-H3K27me3 and anti-survivin antibodies. Anti-Hif1a used to prove the hypoxic state had been induced, and anti-tubulin was used as a loading control. (B–D) Quantification of immunoblots represented in A from three independent experiments demonstrating that EZH2, H3K27me3 and survivin are all more abundant under hypoxia. Data presented are means±s.d. * P <0.05, ** P <0.01, *** P <0.001 (two-way ANOVA with Tukey's multiple comparisons post test).
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    ATCC ela crm ccl 2 cells
    Hypoxia increases expression of EZH2, H3K27me3 and survivin. (A) Immunoblots of WCEs <t>from</t> <t>U2OS,</t> <t>HeLa</t> and MRC5 lines cultured under normoxic or hypoxic environments (24 h). Blots were immunoprobed with anti-EZH2, anti-H3K27me3 and anti-survivin antibodies. Anti-Hif1a used to prove the hypoxic state had been induced, and anti-tubulin was used as a loading control. (B–D) Quantification of immunoblots represented in A from three independent experiments demonstrating that EZH2, H3K27me3 and survivin are all more abundant under hypoxia. Data presented are means±s.d. * P <0.05, ** P <0.01, *** P <0.001 (two-way ANOVA with Tukey's multiple comparisons post test).
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    Hypoxia increases expression of EZH2, H3K27me3 and survivin. (A) Immunoblots of WCEs from U2OS, HeLa and MRC5 lines cultured under normoxic or hypoxic environments (24 h). Blots were immunoprobed with anti-EZH2, anti-H3K27me3 and anti-survivin antibodies. Anti-Hif1a used to prove the hypoxic state had been induced, and anti-tubulin was used as a loading control. (B–D) Quantification of immunoblots represented in A from three independent experiments demonstrating that EZH2, H3K27me3 and survivin are all more abundant under hypoxia. Data presented are means±s.d. * P <0.05, ** P <0.01, *** P <0.001 (two-way ANOVA with Tukey's multiple comparisons post test).

    Journal: Journal of Cell Science

    Article Title: Interplay between nuclear survivin and the PRC2 complex and its impact on H3K27me3-directed transcriptional repression

    doi: 10.1242/jcs.264572

    Figure Lengend Snippet: Hypoxia increases expression of EZH2, H3K27me3 and survivin. (A) Immunoblots of WCEs from U2OS, HeLa and MRC5 lines cultured under normoxic or hypoxic environments (24 h). Blots were immunoprobed with anti-EZH2, anti-H3K27me3 and anti-survivin antibodies. Anti-Hif1a used to prove the hypoxic state had been induced, and anti-tubulin was used as a loading control. (B–D) Quantification of immunoblots represented in A from three independent experiments demonstrating that EZH2, H3K27me3 and survivin are all more abundant under hypoxia. Data presented are means±s.d. * P <0.05, ** P <0.01, *** P <0.001 (two-way ANOVA with Tukey's multiple comparisons post test).

    Article Snippet: Human cervical cancer cells (HeLa), human bone osteosarcoma cells (U2OS), retinal pigment epithelial cells (RPE) and human breast cancer cells (MCF7) were originally from ATCC.

    Techniques: Expressing, Western Blot, Cell Culture, Control